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血凝和纤溶是受激活因子和抑制因子所控制而处于平衡状态的。妊娠和其他情况由于雌激素水平提高,可干扰这种平衡状态引起血液高凝特性,而增加血栓栓塞性并发症。血液高凝特性是因血液凝固因子及血小板的激活,或血凝固抑制因子的下降所致。最重要的抑制因子是抗凝血酶Ⅲ(AT),它能抑制已激活的Ⅻ,Ⅺ,Ⅸ,X和凝血酶的活性。AT的下降可以增加血栓栓塞性并发症的危险。本文作者选择了因伴有或疑有妊娠并发症而住院的96名妊娠后3个月的妇女。根据并发症不同分为:27例子痫前期,10例高血压,4例仅表现胎儿生长迟缓,36例有其它并发症,如心脏疾患,Rh血型
Hemagglutination and fibrinolysis are balanced by activation and repression factors. Pregnancy and other conditions may increase the level of estrogen which may interfere with this equilibrium and cause hypercoagulability in the blood, thereby increasing thromboembolic complications. Blood hypercoagulability is due to the activation of blood clotting factors and platelets, or the decrease of hemagglutination inhibitors. The most important inhibitor is antithrombin III (AT), which inhibits activated Ⅻ, Ⅺ, Ⅸ, X and thrombin activity. The decline in AT increases the risk of thromboembolic complications. The authors selected 96 women 3 months after pregnancy who were hospitalized with or without pregnancy complications. According to the different complications are divided into: 27 cases of preeclampsia, 10 cases of hypertension, 4 cases showed only fetal growth retardation, 36 cases of other complications, such as heart disease, Rh blood group