临床分析及动物实验均报告颅内病变、头部受伤或颅内压增加会导致急性肺水肿 ;在麻醉鼠 ,严重脑部压迫产生急性、猛暴及致死性肺水肿 ,此种神经性肺病变乃由于激发脑干交感神经机构产生体循环高血压及一连串的血流动力学变化 ,导致主动脉血流急剧下降 ,引发肺部血量及血压严重增加 ,最终结果为血管破裂形成出血性肺水肿。 Clinical analysis and animal experiments were reported intracranial lesions, head injury or increased intracranial pressure can lead to acute pulmonary edema; anesthetized rats, severe brain compression resulting in acute, violent and fatal pulmonary edema, such neuropathic pulmonary lesions It is due to stimulate the brain stem sympathetic organs produce systemic hypertension and a series of hemodynamic changes, leading to a sharp decline in aortic blood flow, triggering a serious increase in blood and blood pressure of the lungs, the final result of ruptured blood vessels to form hemorrhagic pulmonary edema.