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一些抗白粉病广谱性有效抗源已在小麦中确定出来，但在许多品种中抗性基因的特性还未知．从１９８２年国际冬小麦白粉病与锈病圃筛选的１０个冬小麦品系作为试材，研究了作用基因的数量与抗白粉病的遗传模式．每个品系都同敏感性品种Ｃｈａｎｃｅｌｌｏｒ杂交，在温室内用１２７号Ｂ．ｇｒａｍｉｎｉｓ小种接种亲本、Ｆ＿１、Ｆ＿２、ＢＣ＿１（Ｃｈａｎｃｅｌｌｏｒ×Ｆ＿１）及Ｆ＿３代群体幼苗，评价了白粉病反应．除过ＳＴ１－２５外，所有亲本都表现抗性。遗传分析表明，Ｃ３９和ＳＩ５中的抗性受３个显性基因控制，Ａ５５－２、Ｒ１０７、ＧＯ４７７９、ＯＫ７５Ｒ３６４５及ＢｕｌｋＰＶ６３－６中的抗性由简单的部分显性基因控制．Ａｒｍａｄａ与Ｃｈａｎｃｅｌｌｏｒ间杂交产生的Ｆ＿２、Ｆ＿３及ＢＣ＿１群体抗性表现不一致，但至少表明Ａｒｍａｄａ有１个抗性基因，很可能是以前指出的Ｐｍ４ｂ．ＶＰＭ１和ＳＴ１－２５中抗性各由１个隐性基因控制．１０个亲本中表现出３至１１个不同的抗性基因．
Some broad-spectrum anti-powdery mildew resistant sources have been identified in wheat, but the nature of the resistance genes in many varieties is unknown. Ten winter wheat lines screened from international winter wheat powdery mildew and rust nurseries were used as test materials to study the number of genes involved and the genetic mode of resistance to powdery mildew. Each line hybridized with the susceptible variety Chancellor, using 127 B in the greenhouse. Graminis races were inoculated with the parents, F_1, F_2, BC_1 (Chancellor × F_1) and F_3 populations to evaluate the powdery mildew response. All parents exhibited resistance except ST1-25. Genetic analysis showed that the resistance in C39 and SI5 was controlled by three dominant genes and the resistance in A55-2, R107, GO4779, OK75R3645 and BulkPV63-6 was controlled by simple, partial dominant genes. The resistance of F_2, F_3 and BC_1 populations produced by crossbreeding between Armada and Chancellor was inconsistent, but at least one resistant gene was found in Armada, probably Pm4b previously indicated. The resistance in VPM1 and ST1-25 is controlled by a recessive gene. Of the 10 parents, 3 to 11 different resistance genes were shown.