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本文介绍利用心肌缺氧模型研究缺氧时在超微结构上的变化,及运用胶体镧作为标志,同时测定灌注液中肌酐磷酸激酶(CPK)含量来观察细胞膜通透性的变化,与对照组相比较观察到的显著变化。现报道如下:
This article describes the use of myocardial hypoxia model changes in the ultrastructure of hypoxia, and the use of colloidal lanthanum as a marker, while perfusion fluid creatinine phosphate kinase (CPK) content to observe changes in cell membrane permeability, and the control group Compared with the observed significant changes. Reported as follows now: