新生儿时期出现黄疸颇为常见。出生前,胎儿分泌的胆红素以非结合胆红素的形式通过胎盘输送给母体,在母体肝脏内成为结合胆红素后由胆汁排泄。出生后,婴儿能在自己的肝脏中形成结合胆红素,但是,几乎每个婴儿的血清胆红素都升高。有时,胆红素血症很严重,应尽量使之成为生理性的,如RH同种免疫很明显的是溶血性疾病表现,但一般较少见。对于大多数婴儿来说,早产通常是加重其黄疸的因素。婴儿出生的越早,很可能黄疸越严重,这在男孩中也比较常见。人们已在数十年前就了解高浓度的血清胆红素能引起脑损伤——核黄疸。核黄疸可以导致死亡,幸存者会产生手足徐动症样的大脑麻痹、精神障碍和神经性耳聋。在换血疗法用 Jaundice in the neonatal period is quite common. Before birth, the bilirubin secreted by the fetus is delivered to the mother’s body through the placenta in the form of unconjugated bilirubin and excreted by the bile after it becomes conjugated to bilirubin in the mother’s liver. After birth, infants can form bound bilirubin in their own liver, but serum bilirubin is elevated in almost every infancy. Sometimes, bilirubin is very serious and should be made as physiological as possible. For RH alloimmunization, it is obvious that hemolytic disease manifests, but it is generally rare. For most babies, premature labor is often a factor in aggravating jaundice. The sooner a baby is born, the more likely the jaundice is, and this is more common among boys. It has been decades since people learned that high concentrations of serum bilirubin cause brain damage - kernicterus. Nuclear jaundice can cause death, and survivors can develop ataxia-like cerebral palsy, mental disorders and neurological deafness. In exchange for blood therapy