连续力竭性运动后,大鼠血乳酸和肌乳酸浓度显著升高,而肌糖原显著降低。虽经48小时休息,血和肌乳酸浓度以及肌糖原均未能恢复至安静水平,肌细胞内线粒体明显水肿,显示乳酸未被有效氧化或合成糖原。这可能与细胞膜的转运障碍及线粒体氧化还原酶系的机能障碍有关。 After continuous exhaustive exercise, blood lactate and lactate concentrations were significantly increased, while muscle glycogen was significantly reduced. After 48 hours’ rest, blood and muscle lactate concentrations and muscle glycogen failed to recover to a resting level, and mitochondria were significantly edematous in muscle cells, indicating that lactic acid was not effectively oxidized or glycogen was synthesized. This may be related to cell membrane transport disorders and mitochondrial oxidoreductase dysfunction.