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1.镁不足对组织的损害 镁可调节腺粒体对钙的摄取,镁不足使钙在腺粒体积蓄,致腺粒体内肌粒病变,进而造成整个腺粒体病变,这与心肌缺血及儿茶酚胺所造成的心肌病变相似。临床及实验证明:致命性缺血性心脏病的首要改变是心肌镁的丢失。猫细胞外镁的耗竭引起缺血后心功能异常及代谢受损。狗膳食镁不足,使冠状动脉闭塞后心肌梗塞的面积扩大。因镁不足而受损的组织中纤维化作用增强和胶原再吸收减慢,因此,镁不足可能是临床
Magnesium deficiency of tissue damage Magnesium can regulate the uptake of calcium in the gland, magnesium deficiency so that calcium in the gland accumulate, cause glandular granulosarcoma lesions, and then cause the entire glandular lesions, which is associated with myocardial ischemia And catecholamines caused by similar myocardial lesions. Clinical and experimental evidence: the first major change in fatal ischemic heart disease is the loss of myocardial magnesium. Depletion of extracellular magnesium in cat cells causes abnormal cardiac function and impaired metabolism after ischemia. Dogs lack of dietary magnesium, myocardial infarction after coronary occlusion area. Fibrosis and collagen reabsorption slowed down in tissues impaired by magnesium deficiency and therefore magnesium deficiency may be clinically