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目的 :探讨肺炎呼吸衰竭合并心力衰竭时酸中毒的阴离子间隙值的变化。方法 :多采用桡动脉血 ,用瑞士产AVL 血气分析仪测定 ,血气及电解质计算阴离子间隙。结果 :呼吸衰竭患儿有阴离子隙增高 ,心力衰竭发生率 72 .7%(16 / 2 2 ) ,阴离子隙不增高仅 2 3.7% (9/ 38) ,二者差异显著。结论 :小儿肺炎呼吸衰竭时代谢性酸中毒及高 AG原因很多 ,血乳酸盐升高致 AG增高占有重要地位。高 AG血症为呼吸衰竭时代谢性酸中毒的一种常见类型。血 AG增高比Pa O2 更能反映组织缺氧。吸氧时 ,Pa O2 常难以反映组织氧化状况 ,此时测定计算 AG是有临床价值的 ,一旦发现血AG明显升高 ,提示组织灌注不良 ,必须警惕 ,及时处理
Objective: To investigate the changes of anion gap of acidosis in respiratory failure patients with pneumonia complicated with heart failure. Methods: The use of radial artery, using Swiss AVL blood gas analyzer, blood gas and electrolyte calculated anion gap. Results: In children with respiratory failure, there was an increase of anion gap, the incidence of heart failure was 72.7% (16/2), and the anion gap was not increased only 2 3.7% (9/38). There was significant difference between the two groups. Conclusion: There are many causes of metabolic acidosis and high AG in children with respiratory failure due to pneumonia. Lactated lactate has an important role in the increase of AG. Hyperagistremia is a common type of metabolic acidosis in respiratory failure. Blood AG increased more than Pa O2 reflect tissue hypoxia. Oxygen, Pa O2 often difficult to reflect the status of tissue oxidation, this time the determination of the calculated AG is of clinical value, once the blood AG was significantly elevated, suggesting poor tissue perfusion, must be vigilant and timely treatment