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局部或全身免疫缺陷常会引起胃肠道并发症。随着治疗进展,患者存活期延长,此等并发症亦显得更为突出。为确定外科对此类病例的作用,本文复习1972年初至1981年底10年内明尼苏达大学医院收治的91位患者。其中33人(36%)共有59项并发症,需手术者24例。91位患者包括7种征群,分属3大类,并发症各有不同,分别讨论如下。第一类:B或T细胞单项缺陷,可表现为三种征群。 1.低丙种球蛋白血症,系B细胞缺陷所致。其IgG、IgA、IgM中一项或多项低于5mg%。本组共41人,内10人共有13项胃肠道并发症。计慢性腹泻或脂肪泻4例;兰伯氏鞭毛虫性肠炎2例;粘膜固有层内结节状淋巴增生1例,出现黑便,须输血1,500 ml;疱
Local or systemic immune deficiency often causes gastrointestinal complications. With the progress of treatment, patients with prolonged survival, these complications also become more prominent. To determine the role of surgery in these cases, we review 91 patients admitted to the University of Minnesota Hospital between the beginning of 1972 and the end of 1981. Of these, 33 (36%) had 59 complications, requiring surgery in 24 cases. 91 patients, including seven kinds of syndromes, belong to three major categories, the complications are different, respectively, discussed below. The first category: B or T cell single defect, can be expressed as three kinds of syndromes. 1. Low gamma globulin, Department of B cells caused by defects. One or more of its IgG, IgA, IgM is less than 5 mg%. A total of 41 people in this group, 10 people within a total of 13 gastrointestinal complications. Chronic diarrhea or steatorrhea diarrhea in 4 cases; 2 cases of Lambertian flagellar enteritis; mucosal lamina propria nodular lymphoid hyperplasia in 1 case, black stools to be transfused 1,500 ml; blisters