长时间缺血可造成组织损伤和细胞死亡。早期再灌注可挽救缺血的心肌组织。但再灌注本身可引起细胞的继续死亡 再灌注损伤。我们利用大鼠缺血再灌注模型来模拟人类心梗后的溶栓过程 ,以了解再灌注损伤与心肌细胞凋亡的关系 ,并加用葛根素进行干预 ,探索葛根素对缺血再灌注损伤的 Prolonged ischemia can cause tissue damage and cell death. Early reperfusion can rescue ischemic myocardial tissue. But reperfusion itself can cause the continued death of cells and reperfusion injury. We used the rat model of ischemia-reperfusion to simulate the thrombolysis after myocardial infarction in order to understand the relationship between reperfusion injury and cardiomyocyte apoptosis, and to add puerarin to intervene to explore the effects of puerarin on ischemia-reperfusion injury. of