革兰氏阳性或阴性菌感染均可引起败血性休克,发病率约占菌血症患者的1/3。临床症状有别于其它型休克,寒战迅速发作于血压下降之前,可处于无末稍血管收缩、体温不低的休克状态。血流动力学改变经历两个阶段;早期末稍血管舒张,张力降低,心输出量正常或增加,中心静脉压常降低,出现呼吸性碱中毒,患者四肢温暖潮红。此 Gram-positive or negative bacterial infections can cause septic shock, the incidence of about 1/3 of patients with bacteremia. Clinical symptoms are different from other types of shock, rapid onset of chills in the blood pressure drop before, can be no end of the vasoconstriction, the temperature is not low state of shock. Hemodynamic changes through two stages; Early end of vasodilation, tension decreased, normal or increased cardiac output, central venous pressure often decreased, respiratory alkalosis, warm limbs in patients with flushing. this