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对神经源性膀胱患儿的临床资料分析中发现,单纯施以逼尿肌加强,尿道肛门括约肌成形术等而未加强盆膈肌的患者症状缓解不明显,而由于神经的损伤或功能障碍,患者的盆膈肌很可能发育不良和松弛,从而加重病人大小便失禁的症状,为证实这一点,作者与临床结合开展了探讨,并总结如下。 1 资料与方法 1.1 20例大小便失禁患儿,病史4～14年,平均8.5年,年龄4～14岁,平均7.5岁,男11例,女9例。1例有骶尾部隐性脊椎裂,19例有腰骶部手术史,其中18例为先天性腰骶部脊膜膨出修补术,1例为骶部畸胎瘤切除术。临床体检20例患儿肛门括约肌均松弛,会阴体下降,低于两坐骨结节连线。
Clinical analysis of children with neurogenic bladder found that simple detrusor enhancement, urethral anal sphincteroplasty and other symptoms without enhanced pelvic diaphragm relief is not obvious, and because of nerve damage or dysfunction, patients The pelvic diaphragm muscle is likely to be poorly developed and loose, thereby exacerbating the symptoms of incontinence in patients. In order to confirm this, the author explored the clinical combination and is summarized below. 1 Materials and Methods 1.1 20 cases of incontinence in children, a history of 4 to 14 years, an average of 8.5 years, aged 4 to 14 years, mean 7.5 years, 11 males and 9 females. One case had sacrococcygeal recessive spondylolysis and 19 cases had a history of lumbosacral surgery. Among them, 18 cases were congenital lumbosacral meningocele repair and 1 case was sacral teratoma resection. Clinical examination of 20 cases of children with anal sphincter relaxation, decreased perineal body, lower than the two ischial tuberosity connection.