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应用单克隆抗体的ＡＰＡＡＰ桥联酶标技术，检测３０例先心病患儿外周血单个核细胞中的Ｔ、Ｂ淋巴细胞及其亚群，旨在细胞亚群水平上探讨先心病易感染的发病机制。结果表明：①先心病患儿存在一定程度的免疫缺陷，尤以青紫组先心病为主；②免疫调节细胞出现异常改变。
Monoclonal antibody APAAP bridging enzyme labeling technique was used to detect T, B lymphocytes and their subpopulations in peripheral blood mononuclear cells of 30 children with CHD. The aim was to explore the pathogenesis of susceptibility to CHD at the level of cell subpopulation mechanism. The results showed that: ① There was a certain degree of immunodeficiency in children with CHD, especially in cyanotic group; ② abnormal changes of immunoregulatory cells.