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对63例确诊的Duchenne肌营养不良症的运动神经传导速度(MCV)和失神经电位的改变进行了统计分析。结果见该类疾患时MCV在尺神经、股神经及腓神经可减慢18.1％～21.4％,与正常人对照组均有显著差异(P< 0.05～0.01)。按年龄分组后的分析可见各组间有显著差异。对这组病例的统计见其有较高的失神经电位发生率(65/428,15.19％),以下肢肌为著。插入电位延长发生率较正常组有增高(17/428.4.0％),重收缩时运动单位电位较正常组亦显著增高。上述改变均提示Duchenne肌营养不良时可以伴有较突出的神经性损害肌电图改变,发生这种情况,分析与进行性肌纤维变性坏死产生的继发性周围神经和运动终板的功能障碍有关。
Statistical analyzes were performed on changes in motor nerve conduction velocity (MCV) and denervation potential in 63 Duchenne muscular dystrophy diagnoses. The results showed that MCV in these disorders in the ulnar nerve, femoral nerve and peroneal nerve can be slowed by 18.1% to 21.4%, and the normal control group were significantly different (P <0.05 ~ 0.01). Analysis by age group shows significant differences among groups. The statistics of this group of patients see its higher incidence of denervation potential (65 / 428,15.19%), to the lower limb muscle. The incidence of prolonged intercalation potentials was higher than that of the normal group (17 / 428.4%), and the motor unit potentials were significantly higher than those of the normal group when the systolic pressure was increased. These changes all suggest Duchenne muscular dystrophy may be accompanied by more prominent neurological damage EMG changes, this situation, analysis and progressive degeneration and necrosis of secondary peripheral nerve and motor end plate dysfunction .