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本實驗比較急性實驗狗、慢性胰瘻狗和經過麻醉的慢性胰屢狗對於鹽酸注入小腸所引起的胰液分泌量和潛伏期,結果證明: (1)在急性實驗情况下,狗胰腺對鹽酸刺激小腸所引起的胰液分泌量遠較在慢性實驗時為少,且潛伏期較長。 (2)巴比妥類麻醉劑:硫賁妥鈉(sodium pentothal)和戊烷巴比妥鈉(sodiumpentobarbital)對鹽酸所引起的胰液分泌量及潛伏期影響極微。 (3)在急性實驗情况下,由鹽酸所引起的胰液分泌量的減少和潛伏期的加長,似乎不是由於巴比妥類麻醉劑的作用,而可能是由於手術創傷的影響。 (4)注射阿托平後,胰腺對於鹽酸刺激小腸所引起的反應顯著减小,故推测在鹽酸引起胰液分泌的機制中可能有神經反射作用的參與。本工作在进行過程中,承蘇聯專家同志親切地給予指導,并承沈(?)淇、劉曾復二教授关懷和支持,(?)此誌謝。
The experimental comparison of acute experimental dogs, chronic pancreatic fistula dogs and chronic pancreatic recurrent chronic pancreatic dogs caused by hydrochloric acid into the small intestine caused by pancreatic secretion and latency, the results show that: (1) in acute experimental conditions, the dog’s pancreas to stimulate the small intestine The induced pancreatic juice secretion is far less than in chronic experiments, and the incubation period is longer. (2) Barbiturates: Sodium pentothal and sodium pentobarbital have a minimal effect on hydrochloric acid induced pancreatic juice secretion and latency. (3) In acute cases, the decrease in pancreatic juice secretion and prolonged latency induced by hydrochloric acid do not appear to be due to the effects of barbiturates but may be due to surgical trauma. (4) After injection of attropine, the response of pancreas to hydrochloric stimulation of the small intestine was significantly reduced, so it is speculated that the mechanism of hydrochloric acid causing pancreatic secretion may be involved in the role of reflex. In the process of carrying out this work, the Soviet expert comrades cordially give guidance and accept the care and support of Professor Shen Ziyu and Professor Liu Zeng Fu II (?).