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Background Arg8]-vasopressin (AVP) activates 3 G-protein–coupled receptors: V1A,V2,and V1B.The AVP-V1A receptor is the primary AVP receptor in the heart； however,its role in cardiac homeostasis is controversial.To better understand AVP-mediated signaling in the heart,we created a transgenic mouse with controlled overexpression of the V1A receptor.