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报道了1例继发于21-羟化酶缺乏症的典型性先天性肾上腺增生(CAH)女婴,其由一位盐份流失型CAH母亲生出。
One case of a typical congenital adrenal hyperplasia (CAH) female infant secondary to 21-hydroxylase deficiency was reported and produced by a salt-losing CAH mother.