暂时性成红细胞减少症(TEC)是婴幼儿一种获得性、自限性、良性疾病,病因尚不明确。但在贫血发生前的2～8周内,常有病毒感染史。目前,尚未研究和鉴定出特异的致病病毒。人类细小病毒(HPV)为遗传性溶血性贫血出现再生障碍危象的一个主要原因。红细胞的生成被抑制可能是HPV对晚幼红细胞直接作用所致。现将1984～1986两年的前瞻性研究结果报道如下。病人和方法全部住院的TEC患儿中3例在1984年12月至1985年3月中旬的第1周期12周内发病,4例在1985年11月下旬至1986年3月中旬的第2周期16周内发病。在相应的两个发病周期内,遗传性 Temporary hypoechongosis (TEC) is an acquired, self-limited, benign disease in infants and young children, the etiology of which is not yet known. However, within 2 to 8 weeks before the onset of anemia, there is often a history of viral infection. At present, no specific virulence has been studied and identified. Human parvovirus (HPV) is a major cause of hereditary hemolytic anemia crisis of aplastic anemia. The inhibition of erythrocyte formation may be caused by the direct effect of HPV on late-age erythrocytes. The prospective findings from 1984 to 1986 are reported below. Patients and Methods All 3 hospitalized patients with TEC developed disease within 12 weeks of cycle 1 in December 1984 to mid-March 1985 and 4 in the second cycle from late November 1985 to mid-March 1986 16 weeks of onset. In the corresponding two incidence cycles, hereditary