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严重烧伤后休克会导致心功能不全,其主要表现为心脏泵机能下降及心肌收缩性降低。近年来通过动物实验和临床研究,逐渐认识到心脏在烧伤休克血流动力学代偿中的作用,除了受一些外在因素影响之外,还受复杂的内在因素控制,即心肌本身长度、张力对产生心肌收缩的影响。本文结合文献综述如下。烧伤休克时心脏泵机能降低的最突出表现之一是心输出量(CO)显著减少。CO为心率和每搏量之积(HR×SV),烧伤后由于血容量锐减,必须通过增快HR来代偿SV不足。狗在三度烧伤面积达50％时HR比对照组增加10～20％,SV减少45％。Shoemaker等观
Severe burn shock can lead to cardiac insufficiency, which is mainly manifested as decreased cardiac pump function and decreased myocardial contractility. In recent years, animal experiments and clinical studies have gradually realized the role of the heart in hemodynamic compensation of burn shock. In addition to being influenced by some external factors, it is also controlled by complicated intrinsic factors, namely the length of the myocardium itself, the tension Impact on the production of myocardial contraction. This article combines the literature is summarized as follows. One of the most prominent manifestations of reduced cardiac pump performance at burn shock is a significant decrease in cardiac output (CO). CO is the product of heart rate and stroke volume (HR × SV), due to the sharp decrease in blood volume after burn, SV deficiency must be compensated by increasing HR. HR in dogs with 50% of third degree burns increased by 10-20% and SV by 45% compared with the control group. Shoemaker et al