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分析经我科诊治的２６０例红细胞葡萄糖６－磷酸脱氢酶（Ｇ－６ＰＤ）缺陷症新生儿高胆红素血症的临床情况。在可查诱因中，以感染占首位（占３９．９％），其次是药物。后者不可忽视来自于孕母或乳母的影响。临床上高胆红素血症的程度轻重悬殊，从生理性黄疸至严重黄疸，甚至发生胆红素脑病。若并存有其他病理状态（如ＡＢＯ溶血病、血红蛋白病等）可加重溶血和黄疸，并使病情复杂化。尚对Ｇ－６ＰＤ缺陷的新生儿易发生高胆红素血症的机理作初步探讨
To analyze the clinical data of 260 cases of neonatal hyperbilirubinemia with erythrocyte glucose 6-phosphate dehydrogenase (G-6PD) deficiency treated by our department. Of the seizure-inducing factors, infection predominates (39.9%), followed by drugs. The latter can not be ignored from the impact of pregnant or nursing mothers. Clinical severity of hyperbilirubinemia severity disproportionate, from physiological jaundice to severe jaundice, and even bilirubin encephalopathy. If combined with other pathological conditions (such as ABO hemolytic disease, hemoglobin disease, etc.) can aggravate hemolysis and jaundice, and complicate the condition. Preliminary Study on the Mechanism of Hyperbilirubinemia in Neonates with G-6PD Deficiency