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实验性近视的形成是眼球主动生长和重新塑形的结果。形觉剥夺或镜片诱导时视网膜释放某些生物活性物质,调节巩膜生长状态和脉络膜厚度,促进近视发生,多巴胺就是其中一种重要物质。本文就多巴胺在眼部的功能,与实验性近视的关系,及其通过多巴胺 D_2受体对实验性近视的作用进行阐述,并就其与其他生物活性物质(褪黑素、乙酰胆碱、全反式维甲酸、成纤维生长因子)之间的相互作用,探讨其对实验性近视的作用机制。
The formation of experimental myopia is the result of active eye growth and remodeling. Form deprivation or lens-induced retinal release of certain bioactive substances, regulating scleral growth and choroidal thickness, and promote myopia, dopamine is one of the important substances. This article describes the relationship between dopamine’s function in the eye and experimental myopia, and its role in experimental myopia through dopamine D 2 receptors. In addition, the effects of dopamine on other biological active substances (melatonin, acetylcholine, all-trans Retinoic acid, fibroblast growth factor) between the interaction between explore its mechanism of experimental myopia.