已知许多药物可加重或诱发重症肌无力,延迟肌肉松弛或对伴有与不伴安全因素已知受损的“正常”人,引起肌无力综合征。包括许多抗生素、抗风湿的,心血管的、抗惊厥和治疗精神病的药物。除了直接作用于神经肌肉内传递的某些药物外,尚有诱导产生自动抗体,抗乙酰胆碱受体蛋白,以及其他作用于脊髓反射或肌纤维本身的药物,因之也增加肌无力。例如苯二氮(艹卓)类,对神经肌肉安全因素降低者特别危险,如前角细胞病变,各种神经病、肌炎、退行性肌病和某些病毒性感染。另一组患者,因罹自动免疫性 Many drugs are known to aggravate or induce myasthenia gravis, delay muscle relaxation, or cause “muscle weakness syndromes” for “normal” individuals with and without known impairment of safety factors. Includes many antibiotics, anti-rheumatic, cardiovascular, anticonvulsant, and psychotropic drugs. In addition to acting directly on neuromuscular delivery of certain drugs, there are also induced autoantibodies, anti-acetylcholine receptor protein, and other drugs acting on the spinal reflex or muscle fiber itself, it also increases myasthenia gravis. Benzodiazepines, for example, are particularly at risk for decreased neuromuscular safety, such as anterior horn cytopathy, various neuropathies, myositis, degenerative myopathies and certain viral infections. Another group of patients, due to autoimmunity