腹水形成的机制腹水形成与以下因素有关: 一、血浆蛋白: 血浆蛋白已认为是肝硬化患者调节腹水的病原学因素之一。在门脉高压情况下,可以预测其毛细管的静水压是增加的,结果引起体液丧失,进入了组织间隙,使组织间隙液体的进入量超过了生理因素的液体回流量,这种液体回流是经过淋巴和组织液胶体渗透压力的;由于组织间隙血浆蛋白的丧失和肠内血浆的去除的结果,使组织液的胶体渗透压更加升高。曾已证实了肝硬化时腹水液系经过肝脏包膜渗出并进入腹膜腔,此液体是起源于肝脏淋巴。肝脏被认为是唯一合成与贮藏白蛋白和组 Mechanism of formation of ascites The formation of ascites is related to the following factors: 1. Plasma proteins: Plasma proteins have been considered as one of the etiologic factors that regulate ascites in cirrhotic patients. In the case of portal hypertension, it is predicted that the capillary hydrostatic pressure is increased, resulting in loss of body fluids, into the tissue space, so that interstitial fluid intake exceeds physiological factors of liquid reflux, the liquid reflux is Through the lymphatic and interstitial fluid colloid osmotic pressure; as a result of the loss of interstitial plasma proteins and intestinal plasma removal, the colloid osmotic pressure of the tissue fluid is further increased. Has proven cirrhosis of the ascites fluid through the liver capsule exudate and into the peritoneal cavity, this fluid is originated in the liver lymph. The liver is considered the only synthetic and storage albumin and group