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本实验选用肝素、地塞米松、布洛芬三种药物，试图增强急性胆道感染大鼠的枯否氏细胞（ＫＣ）补体受体（ＣＲ）功能，结果显示：急性胆道感染（ＡＣ）组、地塞米松处理（ＤＴ）组动物死亡率明显高于肝素处理（ＨＴ）、布洛芬（ＩＴ）和假手术（ＳＯ）组；ＡＣ组术后４８ｈＫＣＣＲ功能持续下降，ＨＴ、ＩＴ能增强感染大鼠的ＫＣＣＲ功能；ＡＣ组肝肺组织细胞明显受损，ＫＣ变性，功能下降，ＨＴ能活化ＫＣＣＲ功能，吞噬活跃，组织受损轻，表明ＫＣＣＲ在急性胆道感染时是机体重要的防御屏障，其功能增强能减少动物死亡和减轻肝肺组织受损。小剂量的肝素是有明显的治疗作用，优于布洛芬和地塞米松。
In this experiment, three drugs, heparin, dexamethasone and ibuprofen, were chosen to enhance the function of complement receptor (CR) in Kupffer cells in acute biliary tract infection. The results showed that acute cholangitis (AC) The animal mortality in dexamethasone treated (DT) group was significantly higher than that in heparin (HT), ibuprofen (IT) and sham operated (SO) groups. KCCR function of rats was significantly inhibited. AC and HCC cells were significantly damaged, KC degeneration, decreased function, HT activated KCCR function, phagocytosis active, damaged tissue light, indicating that KCCR in the acute biliary tract infection is the body’s important defensive barrier, its Enhancement can reduce animal deaths and reduce liver and lung tissue damage. Small doses of heparin have a significant therapeutic effect, superior to ibuprofen and dexamethasone.