肝肾综合征患者可由隐匿性肾功能不全进展到不可逆转的肾功能衰竭,其病理生理特征是肾脏血管继发性收缩致肾脏灌注不良。Bercott及其同事说,内毒素血症促使肾血管收缩学说仍有争议;如果内毒素血症是病因的话,则除了明显的肾功衰竭外,在肝硬化中肾脏灌注不足是可疑的。作者在35例肝硬化病人,经放射性氙~(133) Patients with hepatorenal syndrome may progress from occult renal insufficiency to irreversible renal failure, and their pathophysiologic features are renal perfusion defects secondary to secondary renal vasoconstriction. Bercott and colleagues say that the theory of endotoxemia to promote renal vasoconstriction is still controversial; if endotoxemia is the cause, then, in addition to apparent renal failure, renal insufficiency in cirrhosis is questionable. In 35 patients with cirrhosis, the radioactive xenon ~ (133)