心跳呼吸骤停后血粘滞度增高 ,微血栓形成是影响心肺复苏 (ＣＰＲ)后脑组织再灌注的重要原因。缺血时的组织低氧可使细胞内外环境发生重大变化 ,细胞反应可表现为坏死和凋亡 ,同时细胞也启动修复机制对缺氧损伤进行自我修复[1] 。持续缺血可使脑组织损害加重 ,及时恢复血流有助 Cardiac respiratory arrest increased blood viscosity, microthrombus formation is an important reason for cerebral reperfusion after cardiopulmonary resuscitation (CPR). Ischemic tissue hypoxia can make a significant change in the environment inside and outside the cell, cell response can be expressed as necrosis and apoptosis, while cells also initiate repair mechanisms to repair hypoxia injury [1]. Continuous ischemia can make brain tissue damage, timely recovery of blood flow is helpful