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本实验应用自旋捕捉剂ＰＢＮ证实了开胸麻醉狗与清醒狗心肌顿抑模型的主要区别。冠状动脉阻闭１５ｍｉｎ后再灌注头３小时ＰＢＮ加合物释放总量开胸麻醉狗明显高于清醒狗（约为５倍），而且心肌顿抑的恢复开胸狗明显差于清醒狗。这种区别的主要原因推测是开胸创伤本身以及肾上腺素能神经过度兴奋引起，从而提示在心肌顿抑氧自由基产生的研究中应用清醒动物模型的重要性。
The experimental use of spin traps PBN confirmed thoracic anesthesia dogs and awake dogs myocardial stunning model of the main difference. The total amount of PBN adduct released after 15 min occlusion of the coronary artery in the reperfusion head was significantly higher than that of the awake dog (about 5 times), and the recovery of myocardial stunning was significantly worse than that of the awake dog. The main reason for this difference is speculation that the traumatic thoracic trauma and the adrenergic nerve hyper-excitement are responsible, suggesting the importance of applying a conscious animal model in the study of myocardial stunning with oxygen free radicals.