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巴金森氏病(PD)的乙酰胆硷(Ach)活动明显增加,常被认为是丧失了从黑质传来多巴胺(DA)的结果,但是对丧失这种DA能输入的后果以及由此造成DA—Ach不平衡所涉及的机理还不够了解。
Alzheimer’s disease (PD) has a marked increase in acetylcholine (Ach) activity and is often thought of as a loss of dopamine (DA) delivery from the substantia nigra but results in the loss of this DA and thus The mechanism involved in the imbalance of DA-Ach is not well understood.