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本文报道一例与快速纠正电解质紊乱有关的中央性脑桥髓鞘破坏(CPM),并将CT、核磁共振扫描(NMR),脑电图(EEG)和脑干听觉诱发电位(BAERs)的检查结果进行了比较,以判断BAERs 在CPM 诊断中的作用。病例:女性58岁,甲状腺功能低下病史10年,因感冒后神志模糊、腹泻、呕吐入院。检查示体位性低血压,严重低血钠(97mmol/l),血钾和尿素氮正常,诊断为阿狄森危象,静脉输注氢化可的松和盐水。入院后第3天血钠上升到126mEq/l,但患者出现嗜睡、癫痫大发作,眼球上注视受限,假性球麻痹,四肢轻瘫,腱反射活跃,巴彬斯基征阳性。第5天出现呐吃,病情继续加重,腰穿脑脊液细胞数
This paper reports a case of central pontine myelin sheath destruction (CPM) associated with rapid correction of electrolyte disturbances and examines results of CT, nuclear magnetic resonance (NMR), electroencephalogram (EEG) and brainstem auditory evoked potentials (BAERs) Compared to determine the role of BAERs in the diagnosis of CPM. Case: Female 58 years old, hypothyroidism 10-year history, confusion after flu, diarrhea, vomiting admitted to hospital. Examination showed orthostatic hypotension, severe hyponatremia (97 mmol / l), normal plasma potassium and urea nitrogen, diagnosis of Addison crisis, intravenous infusion of hydrocortisone and saline. On the third day after admission, the serum sodium rose to 126 mEq / l. However, patients developed lethargy, epileptic seizures, restricted attention on the eyeball, pseudobulbar palsy, paresis of the limbs, active reflex tendon, and positive Babski’s sign. Appeared on the 5th day na eat, the condition continues to increase, lumbar cerebrospinal fluid cells