论文部分内容阅读
通过实验性小肠缺血模型，在小肠缺血半小时后恢复灌注，并先应用超氧化物歧化酶进行保护，后再应用谷氨酰胺１周观察大白鼠小肠粘膜湿重，脱氧核糖核酸（ＤＮＡ）含量及肠粘膜形态改变。结果表明，应用谷氨酰胺（ＧＬＮ）后能促进肠粘膜细胞的分裂增殖，加速损伤粘膜的增生、修复。
After experimental intestinal ischemia for half an hour, the perfusion was restored and the superoxide dismutase (SOD) was first used to protect the intestinal mucosa. The glutamine was applied for one week to observe the intestinal mucosa wet weight and the DNA ) Content and intestinal mucosal changes. The results showed that the application of glutamine (GLN) can promote intestinal mucosal cell division and proliferation, accelerate the proliferation of mucosal injury, repair.