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多年来,人们认为碘缺乏是地方性甲状腺肿的基本原因,摄入的碘越少,甲状腺肿的流行率越高。尽管Muarray等报告了,服用药理剂量的碘可引起甲状腺肿;Suzuki也报导了日本北海道渔民,食用海藻引起了碘致的地方性甲状腺肿。但1974年,泛美卫生组织仍以尿碘越少,甲状腺肿越严重,作为评定地方性甲状腺肿病区严重度的标准;1976年,Mitchell等也认为“普通甲状腺肿,实际上是一种碘缺乏的疾病”。因此,迄今国内外绝大多数的地方性甲状腺肿研究工作,几乎都在低碘地区进行。但甲状腺肿在高碘地区和中碘地区的流行率又是怎样?从低碘、中碘到高碘地区甲状腺肿的发病趋势如何?尚缺少明确
Over many years, iodine deficiency is considered to be a basic cause of endemic goitre and the less iodine it ingests, the higher the prevalence of goitre. Although Muarray et al reported that taking pharmacological doses of iodine can cause goiter; Suzuki also reported that fishermen in Hokkaido, Japan, eating seaweed caused by iodine-induced endemic goiter. However, in 1974, PAHO still used less urinary iodine and more severe goitre as a criterion for assessing the severity of endemic goitly warts. In 1976, Mitchell et al. Also considered that “an ordinary goiter is actually a Iodine deficiency disease. ” Therefore, to date, the vast majority of endemic goiter research efforts at home and abroad are conducted in areas with low iodine concentrations. But what is the prevalence of goiter in high-iodine and middle-iodine areas? What is the trend of goiter from low-iodine, middle-iodine to high iodine?