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用气相色谱（ＧＣ）测定了ＰＰ３３３、ＧＡ３处理及未处理苹果果实、果台副梢中内源ＧＡ３、ＧＡ４＋７、ＡＢＡ、ＩＡＡ、Ｚ和ＺＲ的含量，用质谱（ＭＳ）测定了１５Ｎ的分配运转。结果表明，ＰＰ３３３处理明显地增加座果率，抑制果台副梢的增长。而ＧＡ３则具有增大果径和促进果台副梢生长的作用。内源细胞分裂素的含量与苹果着果能力相吻合，其它进素含量的多少与着果没有明显相关。多效唑（ＰＰ３３３）处理明显地提高了内源ＩＡＡ＋ＣＴＫ／ＧＡ４＋７的比值，增加果实中１５Ｎ和全Ｎ的含量。因此可以认为，多效唑提高苹果座果的机理在于减少树体内ＧＡｓ的合成，增加ＣＴＫ的含量，改变树体内的激素平衡，使营养分配由中心果＞果台副梢＞边果变为中心果＞边果＞果台副梢，通过减少果与枝间的矛盾而提高座果。
The contents of endogenous GA3, GA4 + 7, ABA, IAA, Z and ZR in PP333, GA3 treated and untreated apples and apices were measured by gas chromatography (GC). The distribution of 15N was determined by mass spectrometry . The results showed that PP333 treatment significantly increased fruit set rate and inhibited the growth of fruit shoots. While GA3 has the effect of increasing the diameter of fruit and promoting the growth of fruit shoots. The content of endogenous cytokinin is consistent with the apple fruit-fruiting ability, and the content of other elements is not significantly related with the fruit. PP333 treatment significantly increased the ratio of endogenous IAA + CTK / GA4 + 7 and increased the content of 15N and total N in fruits. Therefore, it can be considered that the mechanism of paclobutrazol to increase apple fruit is to reduce the synthesis of GAs in the tree, increase the content of CTK, and change the hormone balance in the tree so that the nutrition distribution changes from the central fruit> the apical fruit> the side fruit to the central fruit> Side fruit> fruit Taiwan Vice shoots, by reducing the contradiction between fruit and branches and improve the fruit.