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多发性硬化 (ＭＳ )病因及发病机制至今未明 ,目前认为它是一种与中枢神经系统髓鞘成分有关的自身免疫性疾病。Ｆａｓ分子是死亡因子Ｆａｓ配体 (ＦａｓＬ )的受体 ,Ｆａｓ/ＦａｓＬ是细胞凋亡的诱导因子[1] ,Ｆａｓ/ＦａｓＬ功能异常可能是自身免疫性疾病的发病机制之一。本文
The etiology and pathogenesis of multiple sclerosis (MS) is unknown so far and is now considered to be an autoimmune disease associated with the myelin component of the central nervous system. Fas molecule is the receptor of Fas ligand (FasL), Fas / FasL is an inducer of apoptosis [1], Fas / FasL dysfunction may be one of the pathogenesis of autoimmune diseases. This article