过去认为重症肌无力是骨胳肌的神经肌肉接头病,故仅累及骨胳肌。近10余年来随着对其发病原理的深入了解,认为本病是由于自身免疫而致乙酰胆碱受体减少所致。故本病是累及全身各部分的胆碱能受体病。因此,有关其内脏症状,诸如心肌无力、吞咽困难、食管无张力、尿失禁、尿频、强迫性尿意和支配瞳孔肌群受累等文献报道日益增多。我院曾诊治1例重症肌无力致动力性梗阻性肾病并发慢性肾功能衰竭(肾衰)的患者,现报告如下: In the past that myasthenia gravis is a skeletal muscle neuromuscular disease, it is only involved in skeletal muscle. Nearly 10 years with the pathogenesis of its in-depth understanding that the disease is due to autoimmune caused by the reduction of acetylcholine receptors. Therefore, the disease is involved in all parts of the cholinergic receptor disease. Therefore, there is a growing literature about the symptoms of visceral disorders such as myocardial weakness, dysphagia, esophageal non-tension, urinary incontinence, urinary frequency, obsessive urinary incontinence, and domination of the pupillary muscles. Our hospital has diagnosed and treated one case of myasthenia gravis in patients with active obstructive nephropathy complicated with chronic renal failure (renal failure), are as follows: