脑血管痉挛(CVS)是蛛网膜下腔出血(SAH)重要的并发症,是引起SAH后脑缺血和脑梗死的病理基础,其发生率30％左右,出现时间多在SAH后3～21天。CVS分为两个阶段:急性痉挛和迟发性痉挛(DCV)。急性痉挛发生在SAH后24小时,常在24小时内缓解。迟发性痉挛不同于急性痉挛,病理解剖学可观察到血管平滑肌细胞发生形态学上的改变,一旦发生很难逆转,对血管扩张剂的反应性差。因此,迟发性痉挛的关键在于预防,有早期发现、早期用药,才能取得一定的疗 Cerebral vasospasm (CVS) is an important complication of subarachnoid hemorrhage (SAH) and is the pathological basis of cerebral ischemia and cerebral infarction after SAH. The incidence of CVS is about 30% . CVS is divided into two phases: acute spasticity and delayed spasticity (DCV). Acute spasm occurs 24 hours after SAH, often within 24 hours to ease. Delayed spasm is different from acute spasm, pathological anatomy vascular smooth muscle cells can be observed morphological changes, in the event of difficult to reverse the vasodilator reactivity poor. Therefore, the key to delayed seizures is prevention, early detection, early medication, to obtain a certain treatment