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一般认为钠潴留在昕有的慢性高血压中起着十分重要的作用,故治疗严重的高血压,都必须严格限制钠盐的摄入。但并非完全如此。在某些病人中可因禁钠而使肾小球滤过率减低,进而增加血尿素氮和肌酐的潴留。某些高血压慢性肾功能衰竭伴有严重失钠,给予盐水溶液时,使严重甚至恶性高血压消退,先兆子病也是一种不宜限钠的高血压,用潴钠降压氯苯甲噻二嗪;疗效较好。
It is generally believed that sodium retention plays a very important role in the occurrence of chronic hypertension in Xin. Therefore, the treatment of severe hypertension must be strictly limited sodium intake. But not exactly. In some patients due to forbidden sodium glomerular filtration rate decreased, thereby increasing blood urea nitrogen and creatinine retention. Some chronic renal failure associated with severe hypertension with sodium, given saline solution, severe and even malignant hypertension subsided, preeclampsia is also a kind of sodium should not be limited to high blood pressure, with sodium chloride hydrochlorothiazide two Triazine; better effect.