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SLE在其临床病程中可能因肾功能减退及使用利尿剂等原因表现出高尿酸血症,但很少并有痛风发作和痛风结节。本文报告1例女性25岁,1976年7月诊断为SLE,内服皮质激素治疗,1979年9月合并肾病综合征,1980年2月发展成慢性肾功能不全,进行一时期的血液透析后予服利尿剂。1982年5月两手指末端出现无自觉症状的皮下结节,逐渐增多,到1984年2月两侧肘关节伸侧、手指、膝盖、足趾也出现皮下结节。深处穿刺时能排出乳白色粘稠样液体。实验室检查血尿酸为12.2mg/dl。组织学检查见真皮下层到皮下脂肪组织有伊红淡染无结构物质形
SLE in its clinical course may be due to renal dysfunction and the use of diuretics and other causes of hyperuricemia, but few and gout attacks and gout nodules. This article reports a female 25 years old, July 1976 diagnosed as SLE, oral administration of corticosteroids, nephrotic syndrome in September 1979 merger, in February 1980 developed into chronic renal insufficiency, after a period of hemodialysis to be served Diuretic. In 1982 May two fingers without symptoms of subcutaneous nodules, gradually increased to February 1984 on both sides of the extensor elbow, fingers, knees, toes also appear subcutaneous nodules. Deep puncture can emit milky viscous liquid sample. Laboratory tests for uric acid 12.2mg / dl. Histological examination of the subcutaneous dermal subcutaneous fat tissue to see there Yiyin no structural material shape