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目的：本文主要探讨新生儿窒息与红细胞增多症的关系及微循环流态变化;方法：对于围产期有窒息史或羊水污染的患儿于入院后24小时做血细胞分析、血气分析及耳廓微循环流态观察;结果：窒息组Hb、HCT、RBC三项值明显高于对照组，85例血气分析27例代谢性酸中毒、10例呼吸性酸中毒、15例混合性酸中毒、6例低氧血症、12例Ⅰ型呼衰、8例Ⅱ型呼衰。45例耳廓微循环观察，综合判断结果：中度异常20例（44.14％），轻度异常18例（40％），大致正常5例（10.2％），正常2例（4.4％）;结论：由于高海拔地区胎儿在宫内即处于低氧状态，慢性缺氧导致代偿性红细胞生成素（RSF）分泌增多，在窒息早期即可表现有局部或全身微循环改变、红细胞聚集、血流缓慢、瘀滞引起组织缺氧酸中毒，严重时导致呼吸衰竭。
Aims: This article mainly discusses the relationship between neonatal asphyxia and polycythemia and changes of microcirculation flow.Methods: The children with asphyxia or amniotic fluid contamination during perinatal period were analyzed on blood cells, blood gas analysis and auricle Results: Asphyxia group, Hb, HCT, RBC three values were significantly higher than the control group, 85 cases of blood gas analysis of 27 cases of metabolic acidosis, respiratory acidosis in 10 cases, mixed acidosis in 15 cases, 6 Cases of hypoxemia, 12 cases of type Ⅰ respiratory failure, 8 cases of type Ⅱ respiratory failure. Forty-five cases of auricle microcirculation were observed. The results showed that 20 cases (44.14%) were moderate abnormality, 18 cases (40%) were mild abnormality, 5 cases were normal (10.2%) and 2 cases were normal (4.4%). : Because of fetal hypoxia in the uterus at high altitude, chronic hypoxia leads to increased secretion of compensatory erythropoietin (RSF), local or systemic microcirculation may manifest in the early asphyxia, red blood cell aggregation, blood flow Slow, stasis caused by hypoxia acidosis, severe respiratory failure caused.