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肺对不同原因损伤的反应形式是一致的。最初为渗出期(24～96小时):有肺泡和肺间质水肿、毛细血管充血、肺泡Ⅰ型细胞结构破坏以及早期透明膜形成。在肺毛细血管中,还常见到纤维蛋白和血小板微血栓以及白细胞聚集。随后为细胞增殖期(3～10天):肺泡Ⅱ型细胞增殖、肺泡隔有白细胞、浆细胞、成纤维细胞和组织细胞浸润;透明膜被成纤维细胞、巨噬细胞和炎症细胞所机化。纤维增殖期;见于创伤后7～10天;肺泡隔和透明膜都有纤维性改变。纤维化最严重的部位是肺泡导管和呼吸性细支气
The response of the lung to different causes of injury is consistent. Initially for the exudative period (24 to 96 hours): alveolar and interstitial lung edema, capillary congestion, alveolar type I cell damage and early formation of transparent membranes. In the pulmonary capillaries, fibrin and platelet microthrombi and also leukocyte accumulation are common. Followed by cell proliferation (3 to 10 days): alveolar type II cell proliferation, alveolar septum with leukocytes, plasma cells, fibroblasts and tissue infiltration; transparent membrane fibroblasts, macrophages and inflammatory cells of the host . Fibrillation; seen in trauma 7 to 10 days; alveolar septa and transparent membrane have fibrous changes. The most severe sites of fibrosis are alveolar ducts and respiratory bronchiolitis