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了解新生儿G-6-PD缺陷症合并巨细胞病毒感染的情况。方法:对新生儿高胆红素血症患儿进行G-6-PD活性、PCR HCMV及其他相关检查。结果:新生儿G-6-PD缺陷症病人合并HCMV（+）者其ALT、IBIL、DBIL、血红蛋白、胆汁酸、G-6-PD活性与新生儿G-6-PD缺陷症HCMV（-）者、非C-6-PD缺陷症HCMV（+）者、非G-6-PD缺陷症HCMV（-）者比较差异均有显著性。结论:新生儿G-6-PD缺陷症合并巨细胞病毒感染会加重肝脏、血液系统的损害,巨细胞病毒感染可降低新生儿G-6-PD活性。
To understand neonatal G-6-PD deficiency combined with cytomegalovirus infection. Methods: G-6-PD activity, PCR HCMV and other related tests were performed in neonates with hyperbilirubinemia. Results: The activity of ALT, IBIL, DBIL, hemoglobin, bile acid and G-6-PD in neonates with G-6-PD deficiency and HCMV , Non-C-6-PD defective HCMV (+), non-G-6-PD defective HCMV (-) were significantly different. Conclusion: Neonatal G-6-PD deficiency combined with cytomegalovirus infection aggravates liver and blood system damage. Cytomegalovirus infection can reduce G-6-PD activity in neonates.