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徐××,男,11岁,于1976年3月患急性黄疸型肝炎住某院。当时SGPT344单位。治疗后仍波动于200单位左右。于6月底(肝炎后三个月)发现患儿血象中白细胞降至2000～3000/立方毫米,中性细胞为60%,红细胞268/万立方毫米,网织细胞0.1%,血小板8.2万/立方毫米,故转来我院。入院时无黄疸,肝脏肿大肋下1.5厘米,剑突下3.5厘米,SGPT330单位,麝香草酚及锌浊度试验正常,HBsAg(-)。骨髓象检查晚幼粒2%,中性带状8%,各系细胞增生减退。入院后给保肝疗法同时给升白细胞药物如沙肝醇,利血生,鹅血片等。并给辅酶A,脱氧核糖核酸,黄芪针剂。患儿入院时舌质淡、嫩、紫,苔薄白,脉虚弦,中医辨证有气血二虚,气虚血瘀,脾肾二虚。故给补气补血温肾中药,并用鲜胎盘口服,必要时输血。有感
Xu × ×, male, 11 years old, was hospitalized with acute jaundice hepatitis in March 1976. At that time SGPT344 units. Still fluctuating around 200 units after treatment. At the end of June (three months after hepatitis), the number of leukocytes in blood was reduced to 2000-3000 per cubic millimeter, 60% of neutral cells, 268 / million cubic mm of erythrocytes, 0.1% of reticulocytes and 82,000 of platelets Mm, so turn to our hospital. No jaundice on admission, liver enlargement rib 1.5 cm, xiphoid 3.5 cm, SGPT 330 units, thymol and zinc turbidity test normal, HBsAg (-). Bone marrow imaging as late juvenile 2%, 8% of the neutral band, the Department of cell proliferation decreased. After admission to the liver therapy at the same time to enhance leukocyte drugs such as calcitriol, Lee blood, goose blood films. And to Coenzyme A, DNA, Astragalus injection. Children admitted to hospital pale tongue, tender, purple, thin white fur, pulse Xuxian, traditional Chinese medicine syndrome qi and blood deficiency, qi deficiency and blood stasis, spleen and kidney deficiency. Therefore, qi and blood Warming kidney medicine, and oral use of fresh placenta, if necessary, blood transfusions. Feeling