本文探讨了100例声门闭合不良的发病机理,其中由于变音期障碍、沟状声带、功能过强性发音障碍,甲状腺功能亢进、感染,神经系统障碍和脑外伤、甲状腺术后和喉外伤者共39例,其余61例原因不明。这61例一侧或双侧的喉内肌CT(环甲肌)、TA(甲杓肌)、PcA(环杓后肌)同时或分别出现了电位时限延长或电静息或波形异常,这充分说明支配该肌的神经有不同程度的麻痹,故原因不明的声门闭合不良可以是不同程度的喉神经麻痹所致. This article explored the pathogenesis of 100 cases of poor glottic closure due to varactory speech disorders, dural vocal cord, dysfunctionalized speech disorders, hyperthyroidism, infections, neurological disorders and traumatic brain injury, thyroid surgery and laryngeal trauma A total of 39 cases, the remaining 61 cases of unknown cause. The 61 cases of unilateral or bilateral laryngeal muscle CT (circumcision), TA (subchondral), PcA (circumflex posterior muscle) appeared at the same time or potential prolongation of electrical potential or electrical rest or waveform abnormalities, which Fully explain the nerves that dominate the muscle have varying degrees of paralysis, so unexplained glottis closure can be due to varying degrees of laryngeal nerve paralysis.