慢性炎性脱髓鞘性多发性神经病(Chronicinflammatory demyelinating polyradiculopathy,CIDP)是一种获得性的周围神经脱髓鞘性疾病,以反复发作的肌无力为特征,可伴感觉缺失和腱反射消失等。其确切病因尚不完全清楚,目前多数学者认为它是由细胞和体液免疫共同介导的,以周围神经髓鞘为攻击目标的自身免疫性疾病[1-3]。皮质激素和静脉滴注免疫球蛋白(IVIG)是最主要的治疗手段,其他传统免疫抑制剂如硫唑嘌呤、 Chronic inflammatory demyelinating polyradiculopathy (CIDP) is an acquired peripheral nerve demyelinating disease characterized by recurrent myasthenia gravis, which may be accompanied by the loss of sensation and the disappearance of tendon reflexes. The exact etiology is not fully understood. At present, most scholars think it is an autoimmune disease mediated by both cellular and humoral immunity and targeting myelinuria [1-3]. Corticosteroids and intravenous immunoglobulin (IVIG) is the most important treatment, other traditional immunosuppressive agents such as azathioprine,