众所周知,肺性脑病是由于慢性肺胸疾患所致的中到重度呼吸衰竭,患者缺氧和有CO_2潴留,并有意识障碍、精神神经症状或体征的一种综合征。其发生机理,主要在于呼吸功能衰竭。由于呼吸功能的衰竭就引起体内高碳酸血症,低氧血症、酸碱平衡失调及脑组织PH值下降等一系列内环境改变。在肺性脑病初期,脑组织改变还多系功能性的;但到中期,脑水肿则开始出现;到后期时,不仅有脑 It is well-known that pulmonary encephalopathy is a syndrome of moderate to severe respiratory failure due to chronic pulmonary-chest disease, hypoxia and retention of CO 2, as well as disturbance of consciousness, neuropsychiatric symptoms or signs. The mechanism of its occurrence, mainly respiratory failure. As a result of respiratory failure caused by hypercapnia, hypoxemia, acid-base balance disorders and brain tissue PH value and a series of changes in the internal environment. In the early stages of pulmonary encephalopathy, changes in brain tissue more functional system; but by the mid-term, brain edema began to appear; to late, not only the brain