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人体内一氧化碳(NO)来源于L-精氨酸并迅速氧化成亚硝酸盐(NO_2)和硝酸盐(NO_3)。NO有扩张血管、调节血小板聚集,神经传导和宿主防御的生理作用。愈来愈多的证据显示,雌激素增加子宫动脉血流量等血管作用可能由NO介导。为研究人类外源性雌激素对NO产生的作用,选择30例平均年龄28岁的育龄志愿妇女,每月注射GnRH类似物De-capeptil3.75mg共两次,4周后以ELISA方法测定血
Carbon monoxide (NO) from the human body originates from L-arginine and is rapidly oxidized to nitrite (NO 2) and nitrate (NO 3). NO has the physiological effects of dilating blood vessels, regulating platelet aggregation, nerve conduction and host defense. More and more evidence shows that estrogen may increase the uterine artery blood flow and other vascular effects may be mediated by NO. In order to study the effect of human exogenous estrogen on NO production, 30 volunteers of childbearing age with average age of 28 years were injected with 3.75 mg of GnRH analogues every month for two times. After 4 weeks, blood samples were measured by ELISA