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肺动脉高压(Pulmonary artery hypertension,PAH)是常见、公认的慢性呼吸系统疾病的并发症之一。在与缺氧性肺病相关的PH中观察到,血管收缩增强、肺血管重塑以及原位血栓形成均促进肺血管抵抗的发展。组织因子途径(tissue factor pathway)调节急、慢性血管损害引起的纤维蛋白沉
Pulmonary arterial hypertension (PAH) is a common and accepted complication of chronic respiratory diseases. It has been observed in hypoxia-associated PH that vasoconstriction, pulmonary vascular remodeling, and orthotopic thrombosis all contribute to the development of pulmonary vascular resistance. Tissue factor pathway regulates fibrin deposition caused by acute and chronic vascular damage