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+ GZ引起急性脑功能障碍是航空医学研究的重要课题之一 ,近年来的基础研究已发现一些重要的基因如 fos、jun、热休克蛋白 - 70和一氧化氮合酶等在反复高 +G值暴露引起脑损伤中起作用 [1 - 3 ] 。但其病理机制还远未明了。是否还存在其它一些相关的、新的基因 ,如何在大范围内同
+ GZ Caused by acute brain dysfunction is one of the important topics in aviation medical research. In recent years, basic research has found that some important genes such as fos, jun, HSP70 and nitric oxide synthase in repeated high + G Value exposure plays a role in brain injury [1-3]. But its pathological mechanism is far from clear. Whether there are other related genes, new, how in a wide range with