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例1:陈×,男,30岁。因畏寒、发热、呕吐伴神志模糊1980年8月入院.T39℃,BP为0,昏睡状,血白细胞重度脱水,呼吸深大,克氏征(-),布氏征(-),22000,中性86%淋巴14%,血钠123mEq/L,血钾6.1mEq/L,CO_2-CP9.1Vol%,脑脊液正常,拟诊为感染性休克。给予扩容(使用含糖溶液)、抗感染、激素等措施后,患者意识障碍加深,血压仍为零,但尿量达300ml/h,尿糖(?),尿酮(?),空腹血糖435mg/dl。停用含糖溶液及激素,给予生理盐水及胰岛素静滴,脱水纠正,血压回升到正常,第3日空腹血糖145mg/dl,改用胰岛素皮下注射,病情稳定,共住
Example 1: Chen ×, male, 30 years old. Due to chills, fever, vomiting with ambiguity admitted in August 1980. T39 ℃, BP 0, drowsiness, severe dehydration of white blood cells, deep breathing, Klebsia syndrome (-), Brinell’s sign (-), 22000 , Neutral 86% lymphatic 14%, serum sodium 123mEq / L, serum potassium 6.1mEq / L, CO_2-CP9.1Vol%, normal cerebrospinal fluid, to be diagnosed as septic shock. After the measures of dilatation (using sugar solution), anti-infection and hormones, the patient’s consciousness is deepened and the blood pressure is still zero, but the urine output reaches 300ml / h, urine sugar, urine ketone and fasting blood sugar 435mg / dl. Disable sugary solution and hormone, given saline and insulin infusion, dehydration correction, blood pressure rose back to normal, the first three days of fasting blood glucose 145mg / dl, switch to insulin subcutaneous injection, stable condition, a total of living