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在前一个实验中,我们进一步证明胰蛋白酶和胃蛋白酶对高蛋白食物具有适应性,并观察到在蛋白酶活性升高的同时,肾上腺重量增加;喂食较长时间后,蛋白酶的诱导适应消失,肾上腺重量的组间差异亦不明显.根据这一现象,我们推想,蛋白酶诱导适应的产生可能必须通过肾上腺的途径.有关垂体一肾上腺系统与消化酶之间的关系虽然已有一些文献报导,如切除垂体引起消化道中腺细胞的退化、酶活性下降,胰腺减重.补充垂体提取液能恢复胰腺重量,提高酶活性;补充肾上腺皮质激素能促进胰脂肪酶的合成等等.但是,在高蛋白食物引起消
In the previous experiment, we further demonstrated that trypsin and pepsin are adaptable to high-protein foods, and observed that adrenal weight increases while protease activity is increased; induction of protease disappears after feeding for a long time, adrenal glands The difference in weight between the groups is also not obvious. Based on this phenomenon, we reasoned that the production of protease-induced adaptation may have to go through the adrenal pathway. The relationship between the pituitary-adrenal system and digestive enzymes has been reported in some literature, such as The pituitary gland causes deterioration of the glandular cells in the digestive tract, decreased enzyme activity, and weight loss of the pancreas. Supplementation of pituitary extract can restore pancreatic weight and increase enzyme activity; supplementation with adrenocorticotropic hormone can promote pancreatic lipase synthesis, etc. However, it is caused by high protein foods. Eliminate