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关于慢性肝炎近年来学者们根据大量病理形态学资料提出:“慢性肝炎是以急性肝炎发展而来、予后是良好的;某中有百分之几经半年以上不能临床治愈者,则称为慢性肝炎。”通过对慢性肝炎(以下简称慢肝)发病机理的研究,认为形成慢肝的主要因素为乙型肝炎病毒(HBV)持续存在和自身免疫反应的结果。现就此两者分别综述如下:HBV 持续存在有报导对220例乙型肝炎表面抗原阳性的急肝患者随访两年结果其中73例(33.2％)乙型肝炎表面抗原(HB_sA_g)仍持续阳性、35例(15.9％)转为迁延性肝炎,10例(4.5％)转为慢性活动性肝炎或肝硬化。而在348例HB_sA_g 阴性的急肝病人中,仅16例(4.7％)转为迁肝、无1例转为慢活肝或肝硬
In recent years, scholars on the basis of a large number of pathological morphological data proposed: “chronic hepatitis is the development of acute hepatitis, to the post is good; a few percent over several months can not be clinically cured, then known as chronic Hepatitis. ”By studying the pathogenesis of chronic hepatitis (hereinafter referred to as“ slow liver ”), the main cause of chronic liver disease is the result of the persistence of hepatitis B virus (HBV) and the autoimmune response. The two are summarized as follows: HBV persistence There were reports of 220 cases of hepatitis B surface antigen-positive acute liver disease were followed up for two years results of which 73 cases (33.2%) hepatitis B surface antigen (HB_sA_g) remained positive 35 Cases (15.9%) into persistent hepatitis, 10 cases (4.5%) to chronic active hepatitis or cirrhosis. Of the 348 HBsA_g-negative patients with acute liver disease, only 16 (4.7%) converted to relocated, and none changed to slow-lived liver or cirrhosis